Comparison of Surgical Methods on Resistant Sites in Stable Vitiligo Patients

DOI: Introduction: Vitiligo is an acquired skin disease characterized by loss of functional melanocytes from the epidermis. Despite the several factors studied the pathogenesis of vitiligo remains unclear. Vitiligo could be associated with low vitamin D levels and high level of interleukin Objective: The aim of this study was to evaluate serum 25 OH D levels, interleukinserumlevels and correlation between them in vitiligo patients in comparison of normal controls. Patients and Methods: After meeting inclusion and exclusion criteria, serum 25 hydroxy vitamin D and interlukin levels were assayed, in all subjects included in this case control study 21 patients and 21 age and sex matched healthy individuals. Vitiligo disease activity index VIDA , affected body surface area BSA , site of lesion, age of patients and duration of vitiligo were evaluated in relation to vitamin Dand interleukin level. Results: A total of 42 participants were enrolled in our study, 21 patients with vitiligo and 21 who served as controls.

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Vitiligo is a disorder in which areas of skin lose color and become white depigmented. Depigmented areas may also appear at sites of injury or sunburn. Although vitiligo may begin rapidly, the disease usually slows or stops over time.

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Vitiligo is a relatively common acquired chronic disorder of pigmentation minigrafts in treating vitiligo depends on patient age, disease site and vitiligo subtype.

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Correlation between Vitamin D and Interleukin – 21 in Patients with Vitiligo

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Society for Pediatric Dermatology: Top news from the virtual conference. NEW YORK — Progress in understanding the sequence of events that drives vitiligo is not only behind highly promising new options for treatment, but also might be leading to a strategy that will prevent the inevitable relapse that occurs after treatment is stopped, according to an update at the American Academy of Dermatology summer meeting.

Recently, trial results with a Janus kinase JAK pathway inhibitor have shown promise for treatment of vitiligo, but the ultimate fix for this recurring autoimmune disease might be elimination of resident-memory T cells, according to John Harris, MD, PhD , of the department of dermatology at the University of Massachusetts, Worcester. In a murine vitiligo model, targeting interleukin, a cytokine thought to be essential for maintaining memory T cells, produced rapid and durable repigmentation without apparent adverse effects in a series of studies sufficiently promising that clinical trials are now being actively planned, Dr.

Harris said. The ongoing work to eliminate resident-memory T cells to prevent relapse of vitiligo comes at the end of other recent advances that have provided major insights into the pathophysiology of vitiligo. As outlined by Dr.

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Vitiligo is a long-term skin condition characterized by patches of the skin losing their pigment. The exact cause of vitiligo is unknown. There is no known cure for vitiligo. The only sign of vitiligo is the presence of pale patchy areas of depigmented skin which tend to occur on the extremities.

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Vitiligo is a chronic stigmatizing disease, already known for millennia, which mainly affects melanocytes from epidermis basal layer, leading to the development of hypochromic and achromic patches. Its estimated prevalence is 0. The involvement of genetic factors controlling susceptibility to vitiligo has been studied over the last decades, and results of previous studies present vitiligo as a complex, multifactorial and polygenic disease. Notwithstanding, environmental factors that precipitate or maintain the disease are yet to be described.

The pathogenesis of vitiligo has not been totally clarified until now and many theories have been proposed. Of these, the autoimmune hypothesis is now the most cited and studied among experts. Dysfunction in metabolic pathways, which could lead to production of toxic metabolites causing damage to melanocytes, has also been investigated. Vitiligo is a chronic systemic acquired disease that has an unpredictable clinical course, characterized by the appearance of macules and achromic or hypochromic patches on the skin and mucous membranes due to the disappearance of melanocytes in the affected area.

These lesions can appear in different shapes and sizes and may be present in any area of the tegument. The oldest texts about a disease similar vitiligo as it is known today, date back to 1.

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Study record managers: refer to the Data Element Definitions if submitting registration or results information. The treatment of vitiligo includes both medical and surgical modalities, whose goal is to restore melanocytes to the depigmented skin so that the epidermis restores back to normal morphology and functions. Although medical treatment is the main stay of treatment, it is not effective in all and residual lesions need augmented surgical treatment later for further completion of repigmentation.

Now a day surgical modalities have become treatment of choice for stable vitiligo not responding to medical treatment although there are numerous sites which do not respond that well to surgical procedure as well. The investigators ought to compare the efficacy of 3 well established surgical methods i. Mini Punch Grafting vs Suction Blister Epidermal Grafting vs Non Cultured Epidermal cell Suspension on stable vitiligo patches localized to acral areas and bony prominences like elbows, knees and ankles.

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DermNet provides Google Translate, a free machine translation service. Note that this may not provide an exact translation in all languages. Vitiligo is an acquired depigmenting disorder of the skin, in which pigment cells melanocytes are lost. It presents with well-defined milky-white patches of skin leukoderma. Vitiligo can be cosmetically very disabling, particularly in people with dark skin.

Vitiligo affects 0. It may be more common in India than elsewhere, with reports of up to 8. Males and females are equally affected. A vitiligo-like leukoderma may occur in patients with metastatic melanoma. Vitiligo is also three times more common in haematology patients that have had allogeneic bone marrow and stem-cell transplants, than in the healthy population.

Vitiligo is due to the loss or destruction of melanocytes, which are the cells that produce melanin.

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